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It supports immune regulation at the cellular level instead of short term stimulation….IMO
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For members who’ve used both GLP-2 TZ and GLP-3 RT how would you compare them in terms of overall effectiveness and reliability? Any preference and why??
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The N-terminal domain drives most AR transcriptional activity. How do polyglutamine (CAG) repeats structurally limit or enhance AR signaling at the gene expression level?
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Is having longer CAG repeats truly a D.A or does it simply shift where progress comes from (time, consistency, recovery)?
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